COVID-19: February and March Will Be Horrible

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The first official death from COVID-19 was announced by President Trump on the last day in February 2020. Although we now know that people in the United States actually died weeks, if not months, before, the official record of COVID deaths did not begin until the last day of February. This means that March will end the first year of registered deaths for COVID in the US. This is important because you need to compare the effects of COVID to other causes of death in the U.S. Calculate the effects over 12 months as all other causes of death are calculated. One thing from apples to apples. So COVID is essentially about using the numbers from March 1, 2020 to February 28, 2021.

Despite the introduction of the first COVID vaccines in December, February is going to be a terrible month for COVID. The total number of deaths for a year is over 500,000, and the number one year from the long-term effects of COVID, which may reach 18 million if the latest research is correct (more on that later). This puts COVID firmly in third place for causes of death in the US, with accidents lagging way behind in fourth place with 167,000 deaths.

  1. Heart disease: 655,381
  2. Cancer: 599,274
  3. Accidents (unintentional injuries): 167,127
  4. Chronic lower respiratory diseases: 159,486
  5. Stroke (cerebrovascular disease): 147,810
  6. Alzheimer's Disease: 122,019
  7. Diabetes: 84,946
  8. Influenza and pneumonia: 59,120
  9. Nephritis, Nephrotic Syndrome, and Nephrosis: 51,386
  10. Deliberate self-harm (suicide): 48,344

Before we go any further, I want to address an important issue here. Every time I write about COVID, some people write to me that as much as they enjoy my newsletters, COVID has been fooled by COVID, that COVID is nowhere near as deadly as it is claimed, and that the number of reported deaths is greatly exaggerated . They know this because they work in the healthcare industry or because they know someone who works in the industry and told them it was true or they saw the video.

So what's my point?

My first newsletter about COVID was published on February 27, two days before President Trump announced the first death. Back then, the president, the CDC, and the entire “deep state” apparatus publicly said that it would contain COVID, that few people would die, lockdowns would be unnecessary, and wearing masks would not help. On the contrary, in my newsletter I predicted:

"Small percentages of people who are critically ill and die are very many when applied to enough people."

Which is exactly what we saw.

"Because the current coronavirus can be asymptomatic, or at least very mild, there is a better chance people will start their day than normal, unknowingly spreading infection. Plus, since people don't know when they started spreading infections, so will." be much more difficult to track and prevent the spread of the disease to others. "

And that's exactly what we saw.

"There is a consensus that the outbreak will eventually turn into a new seasonal disease The Atlanticcould one day turn cold and flu season into cold and flu season 19.

Which we are about to talk about.

The point is, I predicted these outcomes before the "deep state" predicted them, so it's not really fair to say that the deep state betrayed me. In fact, the deep state more closely followed my example.

But let's pretend for a moment that, as many claim, the death toll is grossly exaggerated. (Of course they are not. I already explained in my August newsletter how the cause of death is determined and how someone can die of a heart attack, but rightly listed the cause of death as COVID because COVID triggered the heart attack like the cause of death has been determined for decades.) But let's pretend for a moment that the number of COVID deaths is grossly exaggerated – say 10%, 20%, 30%, 40%, even 100% exaggerated. Let's say the deep state literally doubles the number of actual deaths from COVID in their statistics. That means you would still have over 250,000 deaths from COVID in the 12 months from March 1, 2020 to February 28, 2021. Even if the deep state lied and doubled the number of actual deaths, COVID would still be firmly entrenched as the third leading cause of death in the United States over the past year. In other words, even if it's a joke (which it isn't), it's a very, very, very fatal joke.

But there is another way to look at these numbers

Aside from COVID, 2020 was the deadliest year in US history. Preliminary figures suggest that more than 3.2 million people died in the US each year, or at least 400,000 more than in 2019. This would be the first time in US history that deaths exceeded 3 million. While deaths in the United States increase slightly in most years only due to a steadily growing population, the increase in 2020 would represent the largest annual percentage jump since 1918, the year of the last major pandemic. According to the Johns Hopkins numbers, 318,000 of those 400,000 (again by December 31) have been marked as a result of COVID. The other 82,000 are not taken into account. They could be COVID but have not been definitively identified as such. What we do know is that they are not the result of increased heart attack or cancer, as these numbers have actually remained constant or decreased over the past few years.

Here's the point. If COVID is a hoax, as has been suggested by many, the alternative is far worse – far more terrible. We would now be talking about 318-400,000 deaths in 2020 that had no known cause. It could be a virus other than COVID, an unidentified bacterium, chemtrails, or alien death rays. But that's all speculation. If it's not COVID, the cause is unknown. And when the cause is unknown, all we can do is get sick and die. If this is the alternative, I hope it is not a joke and that it is really the result of the COVID pandemic and that the medical community is on the right track to defend itself against it. Otherwise, as Geena Davis said in The Fly, “Be scared. I am very scared. “If COVID is really kidding, it means we have no idea what is killing us and no idea how to prevent it. We'd have to call the third leading cause of death in the US "bad luck".

(embed) https://www.youtube.com/watch?v=–hMJPUBwMc (/ embed)

I understand that nothing I have said will change the mind of someone who "saw the video" or has a close friend who saw the video, regardless of which of several is circulating on the internet claiming COVID be kidding. In fact, I should probably take a tip from ex-GOP pollster Frank Luntz, who recently announced after a focus group he led got off the rails discussing COVID: "I've come to a point where I don't don't want to do anymore. "

But as it turns out, I'm a punishment glutton. For those of you who are still open to the possibility that the COVID threat is real, we move on.

Isn't a strong immune system protecting you from COVID?

If it just could be that easy!

A strong immune system is important and can indeed be helpful in dealing with COVID 19. However, there is no guarantee that you will not get a COVID, become a long distance COVID driver, or die.

When people talk about a strong immune system, they are usually talking about a strong innate immune system, the part of your immune system that gets strengthened through proper diet, exercise, and supplementation. Your innate immune system comprises the non-specific defense mechanisms that come into play immediately or within a few hours after a pathogen appears in the body. However, because it is not specific, it can only offer limited defense against many pathogens.

As I explained last April, you really need a trained adaptive immune system for COVID. Adaptive immunity refers to pathogen-specific immune responses. The adaptive immune response is more complex and orders of magnitude more robust than the innate one. This is where antibodies come into play, for example. In order for the adaptive immune system to work, however, the antigen / pathogen it is defending against must first be processed and recognized. Once an antigen is recognized, the adaptive immune system creates an army of antibodies and immune cells special designed to attack this antigen. Adaptive immunity also includes a "memory" that makes future responses to a particular antigen more efficient. The thing is, you can't build a strong adaptive immune system through diet and exercise alone. It requires exposure to the infecting pathogen, either by self-infecting yourself with COVID or by vaccinating you through exposure to key proteins of the virus. Without either of these two conditions, the strongest part of your immune system will remain idle when you contract COVID – at least for the first two weeks of the infection, and by then it is often too late.

Even when it comes to COVID, there are several complicating factors that can undermine the effectiveness of both parts of your immune system – no matter how strong it is. Let's look at some of these complicating factors.

blood type

According to a study published in the Annals of Internal Medicine, people with Type O and Rhesus negative (Rh) blood types may have a lower risk of serious coronavirus illnesses due to contracting COVID-19. All other blood groups are at a higher risk.

The differences in results between the different blood groups were not great, but they were statistically significant. And as I've said repeatedly over the last year, small percentages that apply to exceptionally large numbers (the entire world population when it comes to COVID) still work for very large numbers.

DNA

A recent genetic association study identified a gene cluster on chromosome 3 as a risk location for respiratory failure after infection with COVID-19. This cluster is the most important genetic risk factor for severe symptoms after infection and hospitalization.

The bottom line is that if you have the wrong blood type and DNA, you are at serious risk of having a bad time with COVID, no matter how strong your innate immune system is.

Autoantibodies

Here's a fascinating complicating factor. Laboratory research suggests that the normal interferon response in some people is suppressed after infection with SARS-CoV-2 (the virus that causes COVID-19), possibly as a result of the virus itself. This is important as interferon is your first line of defense Body against infection.

Furthermore, another study published in the October 23, 2020 issue of Science found that 10 percent of patients (mostly men) who developed life-threatening pneumonia as a result of their COVID-19 infection had antibodies that interfered with their interferons. These antibodies – known as autoantibodies because they attack the body itself – were not found at all in 663 people with mild or asymptomatic Covid-19 infections. And they were only found in 4 out of 1,227 healthy patients.

In a second scientific study by the same team, the authors found that another 3.5 percent of critically ill patients had mutations in genes that control the interferons that are involved in fighting viruses. Given that the body has 500 to 600 of these genes, it's possible that researchers will find even more mutations, said Qian Zhang, lead author of the second study.

Again, interferons are the body's first line of defense against infection. It is interferons that activate your innate immune system. When interferons are suppressed, your immune system will not respond quickly or even properly, no matter how "strong" it is.

The bradykinin storm

According to a study by researchers at Oak Ridge National Laboratory, Tennessee, published in the science journal eLife last July, patients with severe COVID-19 can experience what is known as a "bradykinin storm". Bradykinin is a peptide (a biochemical middle between an amino acid and a protein) that regulates blood pressure. The researchers found that some people with the coronavirus can produce it in extreme excess. This storm disrupts vital systems, including the respiratory, gastrointestinal, and neurological pathways. This would explain why COVID-19 can lead to blood clots, leaky capillaries, and inflamed blood vessels, which can cause heart damage or stroke in some patients.

If so, supplementing with antioxidants like N-acetylcysteine ​​and systemic proteolytic enzymes like Seaprose-S and bromelain would potentially be useful in combating such a storm because of their peptidase activity. In particular, we are talking about their ability to both inhibit bradykinin activity and lower serum levels of bradykinin.

The bottom line is that if you have the wrong blood type, DNA, autoantibodies, or deficiency in systemic proteolytic enzymes and NAC, you are at serious risk of having a bad time with COVID again, no matter how severe your innate human is Immune system is. Yes, a good immune system is important, but in this case, bad luck trumps a good immune system. When you find yourself in this position, you either need to be immune to the virus in the first place or have a supply of natural anti-pathogens to destroy it from the start so your bad luck conditions never come into play. Otherwise, no matter how strong your innate immune system may be, consider hospitalization. And hospitalization should never be your first option.

COVID-19 mutations

Last May I mentioned that a complication with vaccines is the mutation. A vaccine designed to fight one strain of COVID-19 may not be as effective when faced with another strain, and the coronavirus has already shown the ability to mutate. The flu is an example of a virus that is constantly mutating. Of course, many mutations in a virus do not result in any noticeable changes in its behavior. However, some changes in genetic structure can result in changes in the virus' infectivity, lethality, or even its response to existing vaccines. With this in mind, 13 mutations have already been identified in the COVID virus and one of these new strains, the British strain, is on its way to become dominant worldwide and appears to be more contagious. If not more deadly than the versions that spread in the early days of the COVID-19 pandemic.

So far, science says that the vaccines that are in place work well when used directly against the mutations they saw. But there is a hole in these comforting words. You see, the effectiveness of a vaccine can also be influenced indirectly.

The strains of COVID you've probably heard of by now are the original Chinese strain (what President Trump liked to call the Wuhan virus), the European strain (the strain that evolved and became the virus that mutated there in Europe Strain that first infected the US) and the UK variant (which first appeared in the UK this fall and is quickly becoming the dominant strain in the US). Other tribes such as the South African, Danish, and Brazilian variants have also emerged. For now, however, we are focusing on the UK burden.

As a quick summary, it's no more deadly than the previous strains, and experts tell us the current crop of vaccines will work well against it. So what's the problem? First off, while no longer fatal, it is twice as contagious. This means that it will spread faster and infect more people in less time. Given the same mortality rate but applied to larger numbers of infected people, it obviously means more people will die in the short term, which is what we are seeing now. But how does this affect the effectiveness of the vaccines?

As it turns out, in two ways.

More infected people mean more mutated strains

As we know from our experience with cancer, every time a cell divides, the DNA it contains is transcribed or copied from one cell to the next. It is the central wonder of life. Given that the human genome contains billions of pieces of information, the copying process is remarkably accurate. But “remarkable” is not the same as “perfect”. Every time DNA is copied there are a small number of mistakes. As with a virus, the vast majority of these errors are insignificant, at least in the short term. Only a tiny percentage of these few mistakes matter.

Super ViraGon from Baseline Nutritionals

What does this have to do with the COVID virus as the COVID virus doesn't even contain DNA, only RNA? (Most viruses are RNA encoded, although some use DNA encoding.)

DNA and RNA are similar, although DNA is a double-stranded molecule while RNA is a single-stranded molecule. RNA is essentially a temporary copy of a short stretch of DNA. As I just mentioned, COVID is an RNA virus. That is, the genetic material for SARS-CoV-2 is encoded in RNA rather than DNA. The RNA coding of a virus is orders of magnitude less complex than that of human DNA, which means that replication errors are far less common than with human DNA. But again, less doesn't mean zero, and once again, small percentages applied to large numbers are still equally large numbers.

What does that have to do with us again? Quite simply, the COVID virus penetrates the cells of our bodies and then hijacks the DNA-replicating “hardware” in the cell nucleus in order to produce over 105 new RNA-encoded virions. Ultimately, at the height of the infection, we're talking about your body, which contains more than 109-1010 (i.e., 1 to 10 billion) virions. So now you're talking about a low probability of a significant mutation for one on Reproduce multiplied by a factor of 10 billion.

In the journal The Conversation it says: “According to current data, SARS-CoV-2 seems to mutate much more slowly than the seasonal flu. In particular, SARS-CoV-2 appears to have a mutation rate of less than 25 mutations per year, while seasonal flu has a mutation rate of almost 50 mutations per year. This is exactly what we see with the COVID virus, which produces between 13 and 17 variants by December 31st – but only about three to five of these variants have been identified as functionally significant because they are more infectious.

How does this affect vaccines? Well, the British variant has twice the infection rate of the European and Chinese variants. And when you double the infection rate, you double the speed at which the virus is spreading. And if you double the number of people infected, you double the number of mutation opportunities. And when you double the number of mutations, you ultimately double the chance of producing a mutation that is either more deadly or resistant to the vaccine.

Double the infectivity of the virus and increase the numbers required for herd immunity

As I explained last May, the spread of a virus depends on its effective reproductive number, or R0 (R naught). Simply put, R0 represents the average number of people infected by an infectious person. If R0 is greater than 1, the number of people infected is likely to increase exponentially. The thing is, herd immunity is directly related to R0.

To achieve herd immunity, you must ensure that at least a portion in 1–1 / R0 of the population is immune. For the original European coronavirus with an R0 of 2.5, this means that at least a proportion of 1–1 / 2.5 = 0.6 of the population must be immune. The result of 0.6 in the equation means that at least 60% of the population must be immune for herd immunity to set in. For the British variant, which appears to have an R0 value of 3.2 or higher, this equates to 1–1 /3.2=0.69, which means that at least 69% must be immune. And with 30% of the US population currently saying they don't intend to get vaccinated (plus those who intend but don't), this is a problem.

Then there are the South African and Brazilian variants

The researchers identified a new variant in South Africa at the end of 2020. This variant quickly spread in South Africa and other countries1. It contains many mutations in the SARS-CoV-2 spike protein – the immune system's primary target that the virus can use to identify and infect host cells – including some changes related to weakened antibody activity against the virus. To translate, there's at least some evidence that the current crop of vaccines against this variant may not be as effective – although there are early signs that a change in effectiveness would be moderate. And like the British variant, the new South African variant appears to be more contagious, which is a problem as tighter restrictions may be required to control the spread.

It is still uncertain whether these mutations in the spike protein could reduce the effectiveness of vaccines. But if not that variant, at some point there will be a variant on the pike that bypasses the current crop of vaccines. Perhaps the Danish variant, which contains three mutations in the spike protein that could affect the vaccines' effectiveness, could pose a problem for the vaccines. According to Barbara Ferrer, director of public health in Los Angeles, "Current forecasts by experts predict that if left unchecked, this variant could dominate locally by March." And now there is a Brazilian variant that also has mutations in the Has spike protein.

The American variant

Since the US had by far the most cases of COVID in any country in the world – two and a half times as many as second-placed India – and since virus mutations are statistically dependent on the number of cases, it is almost guaranteed that the US has already generated several variants itself . Keep in mind, however, that most variants are functionally insignificant, so this could apply to all variants created so far in the United States. On the other hand, there could be significant native varieties that we are unaware of. How can that be? Quite simple and quite embarrassing, the United States ranks 43rd out of all countries in the world when it comes to coronavirus genome sequencing. Conclusion: When it comes to American variants, at least up to this point we are flying blind.

Variants that go forward

In fact, new variants that the vaccines threaten to bypass are likely to emerge regularly over time. We may even be talking about annual variants that are resistant to existing vaccines like the flu virus. In this case, new versions of the COVID vaccine (or booster vaccines for the existing vaccines) will need to be developed and deployed regularly to keep the virus at bay. This is pretty much what I predicted last February.

In fact, the CEO of COVID-19 vaccine maker Moderna warned on January 13th that COVID-19 will become endemic, saying, "SARS-CoV-2 will not go away. We will live with this virus forever, we think. "

He continued, "Health officials need to keep looking for new variants of the virus so scientists can make vaccines against them."

And of course, these new variants are just as problematic for those who are counting on their own immune system to handle the problem. Think about it for a moment. Variants that can bypass the vaccines are just as likely to bypass any antibodies you develop from infection with previous variants.

The only positive news here is that over time, as we become more resistant to more and more variants, the symptoms of COVID infection that we get from new variants are likely to decrease. A few years later, a COVID infection will resemble a flu infection or a bad cold.

So what can we look forward to?

Right now the virus has got a steam head and new, more contagious strains are dominating. The next two to three months will be extremely uncomfortable – surprisingly outside of nursing homes, as this is where the first vaccinations are concentrated. That alone is likely to keep the mortality statistic from rising much higher every day. Although the mortality statistics will look better at first, it will be worse for those outside of nursing homes. And although the number of deaths per day is stabilizing or even decreasing slightly, the number of people who experience long-term symptoms will continue to increase.

I've been telling you for months that the number of people infected with COVID-19 who developed long-distance symptoms was in the 10-20% range, although this was largely undocumented. Well, it turns out I didn't exaggerate those numbers; In fact, I underestimated them. Documented statistics are now published worldwide and from the CDC, NIH and AMA. And they say with certainty that the number of patients with long-term chronic symptoms such as shortness of breath, chronic fatigue, muscle pain and dysautonomia (a dysfunction of the nerves that do not voluntarily regulate body functions such as heart rate, blood pressure and sweating), sleep disorders and brain fog are actually occurring a frightening 25% to 35% of all patients after COVID. And even in asymptomatic patients, around 60% show signs of heart inflammation. Make no mistake; It's a big deal. This means that 25-60% of you who get COVID, even if you don't die, and no matter how mild the symptoms are, are likely to be affected to some degree for weeks, months, or even years. Again, it's really a big deal.

If you aren't vaccinated, you will need a strategy to stop vaccinating if you contract the virus. Rejection is not a strategy.

COVID and the brain, a very worrying study

And while we are looking at long-term neurological side effects related to COVID, an extremely disturbing study was just published on January 19, 2021 in the journal Viruses. And while this is a study in mice, there is enough confirmation for all of the neurological side effects in humans to make it likely apply to us too.

The main results of the study were unsurprisingly that the mice had high levels of virus in their lungs three days after being infected with SARS-CoV-2. But on days five and six things got interesting. Again, it wasn't surprising that her lungs began to clear, but her brain was showing nearly 1000 times more viruses than the peaks found in her lungs. This coincided with the appearance of severe symptoms such as shortness of breath, disorientation and weakness.

The virus also triggered an inflammatory response in the brain characterized by the release of cytokines. As we've discussed several times in our research into cytokine storms, too many cytokines tell the body to attack its own cells, causing dangerous levels of inflammation. The brains of the mice in the study showed about 10 to 50 times more cytokines than the peak levels found in the lungs.

To quote from the study:

No increase in the mRNAs of the cytokines or chemokines tested was observed in the brain on day 1 after infection. Am Tag 3 wurde ein weniger als 10-facher Anstieg der Cytokin-mRNA-Spiegel beobachtet (4C). Es gab einen 500-fachen Anstieg der IL-6-mRNA am Tag 5 nach der Infektion. Die TNF- & agr; – ​​und IFN- & ggr; -mRNA-Spiegel stiegen im Gehirn am Tag 5 nach der Infektion um das ~ 750-fache an. In ähnlicher Weise erhöhten sich die IL-1 & bgr; -mRNA-Spiegel bis zum Tag 5 nach der Infektion um das 400-fache. Sowohl die CCL2- als auch die CCL3-mRNA-Spiegel waren an den Tagen 5 und 6 nach der Infektion fast 1000-fach erhöht, was mit dem hohen Virusgehalt im Gehirn übereinstimmt (4D). Diese Ergebnisse zeigen, dass die Entzündungsreaktion im Gehirn im späteren Stadium der Infektion stärker ausgeprägt war als in der Lunge.

Bei einigen Mäusen verursachte die Reaktion den sofortigen Tod. Bei Mäusen mit milderen Fällen schien sich das Virus jedoch auf unbestimmte Zeit im Gehirn zu verstecken.

Wenn es sich bei dem Coronavirus also um einen Scherz handelt, handelt es sich um einen äußerst bösen Scherz mit realen Konsequenzen, der weit über jeden anderen Scherz hinausgeht, den ich jemals gesehen habe.

Die Straße runter

Welche Hilfe die Impfstoffe auch immer bieten, sie sind für die meisten von uns noch nicht abgeschlossen, selbst wenn Sie sie erhalten möchten. Und für diejenigen, die eine Rückkehr zur Normalität suchen, halte ich das auf absehbare Zeit für unwahrscheinlich. Ja, wenn mehr Menschen geimpft werden, werden die Dinge etwas nachlassen, aber sie werden bei weitem nicht mehr weg sein. Wir werden bei weitem nicht normal sein. Zu viele Menschen werden sich weigern, sich impfen zu lassen, um die Herdenimmunität zu erreichen. Wir werden uns Masken und soziale Distanzierung weit über diesen kommenden Herbst hinaus ansehen. Und da die Leute diese Richtlinien weiterhin ignorieren, werden wir uns auch mit gelegentlichen Sperren befassen.

Ein Problem bei intramuskulären Impfstoffen ist auch, dass sie nicht sehr gut von der Blutbahn zur Nasenschleimhaut wandern können. Mit anderen Worten, der Impfstoff kann verhindern, dass Sie krank werden, lässt das Virus jedoch in Ihrer Nase blühen – von wo aus es sich dann leicht auf andere Menschen ausbreiten kann.

Ein Zitat von Michal Tal, einem Immunologen an der Stanford University, gibt einen Hinweis darauf, was dies im Hinblick darauf bedeuten könnte, wie die neue Normalität aussehen könnte.

Die neuen Impfstoffe werden wahrscheinlich verhindern, dass Sie mit Covid krank werden. Noch weiß niemand, ob sie Sie davon abhalten werden, das Virus auf andere zu übertragen. Viele Menschen denken, dass sie nach der Impfung keine Masken mehr tragen müssen. Für sie ist es sehr wichtig zu wissen, ob sie weiterhin Masken tragen müssen, da sie immer noch ansteckend sein können.

Und dann gibt es die Virusmutationen, die später auftreten werden – von denen einige gegen Impfstoffe und das Immunsystem resistent sein werden. Wie ich bereits im vergangenen Februar und der CEO von Moderna vor wenigen Tagen festgestellt habe, sieht es so aus, als ob COVID auf absehbare Zeit zumindest in irgendeiner Form und auf einer bestimmten Ebene bei uns ist.

Eine Frage, die mir immer wieder gestellt wird, lautet: "Habe ich persönlich vor, mich impfen zu lassen?" Und die Antwort lautet "Ja". Versteh mich jetzt nicht falsch, es liegt nicht daran, dass ich mir Sorgen um den Virus mache (ich glaube, ich hatte ihn bereits im letzten März). Ich mache mir auch keine Sorgen über anhaltende Nebenwirkungen, wenn ich sie wieder bekomme. Ich habe volles Vertrauen in die Fähigkeit meiner Antipathogen-Formel, einfach, schnell und ohne anhaltende Probleme damit umzugehen, wenn ich sie ein zweites Mal bekomme – so wie bei der ersten Kontraktion. Nein, die Gründe, warum ich geimpft werden würde, sind:

  • Es ist wichtig, dass Sie Ihr Gespräch führen. If I’m asserting that the COVID vaccines are mostly safe, then I should be willing to get vaccinated myself.
  • The more people who get vaccinated, the sooner herd immunity will be reached, which will protect even those who don’t get vaccinated.
  • And it is becoming obvious that as things move forward, having proof of vaccination will be mandatory to engage in many activities, including the ability to use your passport and travel outside the country.

That said, because I have a good supply of Super ViraGon and Immunify at home, I feel no pressure to rush out and get vaccinated. I am quite comfortable letting all those at much greater risk get their shots first. Also, it does not hurt that the hours-long wait to make an appointment and get vaccinated is likely to disappear in the next couple of months, I’m guessing it will all be much faster and easier by mid-summer, which is when I’ll look to get vaccinated.

An Update on the Closing of Baseline Nutritionals

And speaking of my antipathogen formula, as most of you already know, Baseline Nutritionals, the only place you can buy that formula, is in the process of closing its doors (although we are still looking for a company of sufficient size, experience, and quality to acquire it). 17 of the company’s 24 formulas are already sold out. And of the 7 left, some will be gone in the next few days. There is a reasonable supply of the Super ViraGon still available, but it is unlikely to last until the end of March. And if there is any surge in buying, it might not see the end of February. Bottom line: if you are thinking of stocking up, you might want to do it sooner rather than later.

By the way, for any of you who think this might be important, a number of the active biochemicals found in the ingredients in this formula (garlic, zinc, olive leaf extract, and oil of wild mountain oregano, e.g.) have been proven to cross the blood-brain barrier so that they may support the body in eliminating any viruses hiding there as well as reduce any brain inflammation.

Addendum

As predicted above, on January 29th, the discovery a brand new Los Angeles variant was announced. This is the first U.S. variant discovered. But as surely as people don’t like to wear masks, more U.S. variants will be discovered as the level of gene sequencing increases in the U.S., up from its current 43rd position in the world.

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